Denosumab — Drug Monograph

Brand names: Xgeva (oncology — 120 mg SC q4w), Prolia (osteoporosis — 60 mg SC q6m; different product — do NOT interchange)

Drug class: Supportive Care Agent

Mechanism of Action

Fully human IgG2 monoclonal antibody that specifically binds RANKL (receptor activator of nuclear factor kappa-B ligand) with high affinity, blocking its interaction with RANK on osteoclast precursors. RANKL is expressed by osteoblasts, bone marrow stromal cells, and tumor cells in the bone microenvironment; it is the principal mediator of osteoclastogenesis, osteoclast activation, and osteoclast survival. By neutralizing RANKL, denosumab prevents osteoclast differentiation from precursors and induces apoptosis of mature osteoclasts, profoundly suppressing bone resorption. Compared to…

FDA Indications

Common Side Effects

Clinical Pearl

Denosumab's key clinical advantage over zoledronic acid is that it requires NO renal dose adjustment, making it the preferred bone-protective agent in patients with CKD (CrCl <30–60 mL/min) and in multiple myeloma (where renal impairment is common). However, hypocalcemia is more frequent and can be more severe than with bisphosphonates — calcium and vitamin D supplementation is mandatory, not optional. When stopping denosumab after long-term use (especially in adolescents/GCT patients), always…

Related Therapies & Mechanisms