selinexor — Drug Monograph

Brand names: Xpovio

Drug class: Other

Mechanism of Action

Selinexor is a first-in-class selective inhibitor of nuclear export (SINE) that covalently binds to XPO1 (exportin-1, CRM1), the primary nuclear export receptor responsible for shuttling tumor suppressor proteins out of the nucleus. Inhibition of XPO1 causes nuclear accumulation and functional reactivation of key tumor suppressors including p53, FOXO1/3, IκB, RB1, and CDKN1A (p21), as well as reduction of nuclear export of oncogenic mRNAs (including MYC, MDM2, BCL-2 mRNAs). This results in cell cycle arrest and apoptosis selectively in cancer cells with aberrant XPO1-dependent export activity.

FDA Indications

Common Side Effects

Clinical Pearl

GI toxicity with selinexor is predictable and largely manageable with aggressive prophylaxis — the standard of care includes a 5-HT3 antagonist (ondansetron 8 mg) plus dexamethasone 8 mg taken 30–60 minutes before every selinexor dose, with an NK1 antagonist added for higher-dose regimens. Hyponatremia occurs in ~30–40% of patients and is thought to be a CNS-mediated syndrome of inappropriate antidiuretic hormone (SIADH) effect; sodium supplementation or fluid restriction may be required. The…

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